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What is the primary mechanism of action of cyanide toxicity in the body
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What is the primary mechanism of action of cyanide toxicity in the body? • Cyanide toxicity poses a grave threat to the human body, disrupting cellular respiration and leading to severe metabolic dysfunction. When cyanide enters the body, it binds rapidly to cytochrome c oxidase, a key enzyme in the electron transport chain of mitochondria, inhibiting aerobic metabolism and halting ATP production. This interference deprives cells of essential energy, resulting in tissue hypoxia and metabolic acidosis. Symptoms of cyanide poisoning can manifest rapidly and include headache, dizziness, nausea, and ultimately progress to seizures, respiratory failure, and cardiac arrest if left untreated. Swift recognition and intervention are critical in managing cyanide toxicity, typically involving administration of antidotes such as hydroxocobalamin or sodium thiosulfate to facilitate detoxification. Understanding the mechanisms and consequences of cyanide toxicity is paramount in both medical emergency response and forensic investigation. • #CyanideToxicity #ChemicalSafety #MedicalEmergencies #Toxicology #Healthcare #Biochemistry #EmergencyMedicine #ChemicalPoisoning #PublicHealth #ClinicalChemistry • #ClinicalChemistry • #MedicalLab • #BiochemicalAnalysis • #LabMedicine • #DiagnosticTests • #Biomarkers • #HealthcareScience • #LabTechniques • #MedicalTesting • #ChemicalAnalysis
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