Heart Failure and RASS Renin angiotensin Aldosterone system 3D animation

>> YOUR LINK HERE: ___ http://youtube.com/watch?v=RoV9sKBpYaY

In this video, we have shown animated view of heart and #heart #failure and explained renin angiotensin system or #RAAS in details. • Despite ongoing advances in the management of chronic heart failure, patient outcomes for acutely decompensated disease have not improved. Across Canada, patients hospitalised for heart failure continue to suffer and offer frequent complications, and 16. 5% die during their initial hospitalisation. An examination of the pathophysiology of acute decompensated heart failure identifies promising new avenues for treatment. During heart failure, the heart cannot circulate enough blood of normal cardiac filling pressures to meet the metabolic needs of the brain, kidneys, heart, and other vital organs. The body responds by releasing neurohormones. Certain neurohormones help maintain blood pressure, but over time they cause heart failure to worsen. One of these neurohormones is renin released by the kidneys in response to decrease the fusion and increased sympathetic activity. • Renin activates the renin angiotensin aldosterone system, or Raas, by cleaving angiotensinogen in the liver to produce angiotensin I, which is further converted to angiotensin II. In the lungs, angiotensin II binds to blood vessel walls, causing vasoconstriction. Angiotensin II also stimulates release of endothelium from endothelial cells. Angiotensin II and endothelium act on vascular smooth muscle, causing constriction of the coronary arteries and systemic arteries and veins. While vasoconstriction helps maintain blood pressure, it can also have maladaptive effects. for example, the weakened heart must pump harder against increased resistance in the systemic arteries, while constriction of the coronary arteries further compromises the myocardium supply of oxygen.

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